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To support less frequent administration, site-specific lipidation or PEGylation was performed to prolong the duration of action of the unimolecular dual incretins

It works by binding to ghrelin receptors (GHS-R) in the pituitary, prompting the release of GH

A second mechanism of PKA-independent incretin potentiation of glucose-stimulated insulin secretion (GSIS) involves cAMP-regulated guanine nucleotide exchange factor (cAMP-GEF) EPAC2

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No serious adverse events have been reported across extensive research use