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In vitro experiments confirmed that A 25-35 treatment significantly inhibited the activation of the NRF2/SLC7A11/GPX4 signaling pathway in HT22 cells, upregulated the expression of Fe 2+ , TFR1, ACSL4, MDA, and GSSG, and decreased the expression of FTH1, SOD, and GSH, as well as the GSH/GSSG ratio, resulting in dysregulated neuronal iron metabolism, impaired amino acid antioxidant systems, mitochondrial dysfunction, and ferroptosis, which ultimately promoted neuronal injury

Excessive ROS damage to vascular endothelial cells disrupts the balance between vasoconstrictive and vasodilatory factors, impairing the hemostatic contractility of spiral arteries

[PMID: 2272034] PubMed record related to this compound for laboratory literature review

This 15-amino acid peptide, derived from gastric protective protein, works by stabilizing cellular membranes and promoting angiogenesis while simultaneously reducing inflammatory cascade activation

Palmitoyl-L-carnitine induces Tau phosphorylation and mitochondrial dysfunction in neuronal cells

Characteristics of PFIC and PFIC-related diseases in paediatric populations PFIC causes progressive intrahepatic cholestasis in newborns, infants, and children