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(PubMed) Harris JC, Cottrell SL, Plummer S, Lloyd D
PMC 2438277
Thus, we may assume that increased levels of all studied genes could be a result of the glutathione depletion triggered by UA application
10.1371/journal.pone.0006413 PLoS One
PHGDH-mediated endothelial metabolism drives glioblastoma resistance to chimeric antigen receptor T cell immunotherapy
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