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At t = 4 h, cells were serum starved and at t = 1 h, pre-treated with vehicle, control IgG1, or L2-Cmu (100 g/mL)

By inhibiting the reuptake of these neurotransmitters, they are more present around cells

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GIP-induced inhibition of centrally mediated emesis signals triggered by GLP-1 administration enhances weight loss seen with GLP-1 RA administration