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We took a straightforward approach: source pharmaceutical-grade reduced glutathione and dose it at clinically relevant levels

We hypothesize that reduced CPT-I activity may contribute to persistent fatigue in these congenital hypothyroid patients, even after levothyroxine therapy, which alone may be insufficient to fully address the downstream metabolic consequences of TPO and TSHR gene mutations present in this cohort

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However, pregnancy losses and terminations remain elevated in populations with obesity and T2DM, reflecting underlying maternal risk