There are also limitations to the study from the Mayo Clinic researchers
White WB, Bakris GL, Bergenstal RM, Cannon CP, Cushman WC, Fleck P, Heller S, Mehta C, Nissen SE, Perez A, Wilson C, Zannad F
The observed changes in GLP-1 level following low FODMAP diet intake might be explained by chemosensory mechanisms in enteroendocrine cells involved in food intake-mediated changes in intestinal hormone levels
GLP-1 receptor agonists have known satiety-promoting effects due to central action of the neuropeptide GLP-1 [1], with reduced activation of appetite- and reward-related brain areas in response to food [18]
Effects from animal studies and reports may start in days for acute issues, up to weeks for chronic repair
In the lifestyle counseling alone group, changes were +0.01 ng/mL for CTX (95% CI: 0.05 to 0.07) and 0.58 mg/mL for P1NP (95% CI: 6.69 to 5.52)]