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We tested therefore also mutants impaired in the three genes SULTR1;2 , SULTR4;1 and SULTR4;2 encoding sulfate transporters 64,65,66 but did not observe an altered susceptibility to AITC

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These in vivo results align with the in vitro data, consolidating the understanding that GHK-Cu exerts its protective effects in ALI by inhibiting excessive inflammatory responses through its anti-inflammatory and antioxidant properties, mediated by the suppression of NF-B and p38 MAPK signaling pathways [2]

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