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These structural and functional observations have been integrated under the broader concept of thalamocortical dysrhythmia, in which persistent alterations in thalamic output and thalamocortical rhythms are thought to contribute to interictal features of migraine such as deficient habituation and multisensory hypersensitivity [35]
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GPX4 activity (levels of 15-HpETE-PE and 15-HETE-PE) was not directly measured in relation to inflammatory marker expression and cell death
