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A comprehensive bioinformatic analysis was conducted, incorporating GLP1R, Gastric Inhibitory Polypeptide Receptor (GIPR), Gastric Inhibitory Polypeptide (GIP) and glucagon receptor (GCGR) to assess the effects of GLP1R agonists on gene and metabolite interactions

The GIP receptor component may further augment insulin release and support fat metabolism

Altogether, it may possibly induce a steady and enhanced release of GH