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Understanding how peptides work at the receptor level helps explain why dual agonism amplifies single-target effects
In a dedicated review article on the lipid-centric vs protein-centric mechanism of action by anesthetics, Eckenhoff [327] drew examples to the fact that the effects seen on membrane properties were detectable at concentrations far above clinical concentrations, with the caveat that these model cell membranes may be poor approximations to real, highly complex biological systems
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