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Unfortunately, in this study, no analysis with respect to fracture type and involvement of both endplates and extent of endplate damage was done

3.5 GHK-Cu regulated the SIRT1/STAT3 pathway SIRT1, which is closely associated with cellular metabolism and stress responses, also interacts with the STAT3 pathway (Figures 6AD, DSS-induced mice exhibited significantly increased SIRT1 expression and decreased p-STAT3 expression compared to the Control group

While methodological variability exists, the convergence of cellular, enzymatic, and histological endpoints strengthens the evidence supporting biologically meaningful redox modulation by GHK-Cu

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