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Mathes CM, Bueter M, Smith KR, Lutz TA, le Roux CW, Spector AC
(1992, Biochemical Journal) showed IGF-1 LR3 produced larger anabolic responses than native IGF-1 in dexamethasone-induced muscle catabolism in rats
They work by blocking the action of enzyme dipeptidyl peptidase 4 (DPP-4) in order to prevent the rapid degradation of endogenously released GLP-1, thus favoring glucose regulation
Immune System Support
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Effectiveness varies in clinical trials
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