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Inflammatory mediators such as histamine, proteolytic enzymes, glycolytic enzymes, and heparin, as well as de novo synthesized prostaglandin D2 (PGD2), leukotriene C4 (LTC4), adenosine, and ROS are released from mast cell granules (34), and they directly induce oxidative stress ( Figure 1 )

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Similarly, Sp treatment resulted in maintained mitochondrial complex I activity which is found to be disrupted in PD patients and Parkinsonian models (Fig

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claims-based analysis found approximately a 33% lower risk of Alzheimers disease and related dementias among GLP-1 users compared with patients treated with other glucose-lowering therapies [9]