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A reduced initial dose should be considered for patients known to be homozygous for the UGT1A1*28 alleleHowever, the precise dose reduction in this patient population is not known 43 Smith, Phase II Metabolism, UNC Chapel Hill, 8/2012 Other polymorphisms of UGT due to random genetic differences have been reported, but only a few have suggested to influence drug metabolism in the clinic.e.g

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A complex II defect affects mitochondrial structure, leading to ced-3- and ced-4-dependent apoptosis and aging

Arterioscler Thromb Vasc Biol 2007