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The neuronal loss and atrophy were particularly evident in the CA1 region of the hippocampus and layers II-III of the cerebral cortex

Discussion In this study, we demonstrate that METTL3-mediated m 6 A modification functions as a critical regulatory mechanism in the transition from primed to nave pluripotency

PGE(2)-driven induction and maintenance of cancer-associated myeloid-derived suppressor cells

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