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These results suggested that keloid fibroblasts underwent pro-senescence regulation earlier compared with normal skin fibroblasts via p53-pS15 under serum limitation stimulus, and p53-pS15 and FOXO4 translocated from the nucleus to the cytoplasm during FOXO4-DRI treatment

This unbalanced regulation of the glutathione synthetic pathway makes it hard to explain the rapid and steady increase of glutathione abundance in infested resistant plants