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Most studies leverage joint-cavity acidification for targeted delivery of mitochondrial antioxidants, metabolic modulators, or autophagy-promoting molecules to diseased chondrocytes to restore mitochondrial homeostasis

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however, in roots, ABA induced the expression of GhGPX1 and GhGPX13 , while no obvious change was found for GhGPX8

Interestingly, CFS is also associated with low levels of this important antioxidant

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Understanding complementary mechanisms guides effective combination strategies