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K.Prez-RontomC.NavascusJ.et al (2012)
The major key for nephrotoxicity is oxidative stress due to tissue inflammation
Dampened ER stress signaling was also evident from diminishment of the splicing of the IRE1 nuclease target Xbp1 (Figure 1B) and of the upregulation of the stress-regulated factor CHOP (Figure 1C)
Funding Open Access funding provided by The Francis Crick Institute
GA caused multiple forms of cell death, such as apoptosis characterized by mitochondrial cytochrome c release and caspase-3 activation, ferroptosis characterized by lipid peroxidation, and necroptosis characterized by loss of plasma membrane integrity, in an iron-dependent manner (Tang and Cheung, 2019)
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