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For example, exogenous monounsaturated fatty acids (MUFAs) can suppress ferroptotic sensitivity by remodeling membrane phospholipid composition and limiting lipid peroxidation
Thus, a deficiency of GLP-1 is now considered as part of the pathophysiology of type 2 diabetes 1
In summary, QUEs dual, dose-dependent naturecharacterized by protective effects at low doses and toxic effects at high doseshighlights its double-edged sword status
Ex vivo assays of islets isolated from both genotypes demonstrate comparable insulinotropic responses to glucose, as well as enhancement of GLP-1-, GIP-, OXM-, and EX-4-stimulated insulin secretion
a recently reported References Shah Z, Hundal J, Afridi SS, et al
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