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Lycium barbarum polysaccharide counteracts this hepatotoxicity by preserving mitochondrial integrity and activating Nrf2 signaling, supporting its potential as a protective agent against cadmium-induced liver damage (126)

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at the same 1 ng/ml) displayed lower activation activity than the maximum level of the LASSS-treated HGF, aligning with the established HGF-dose-dependent manner of satellite cell activation with maximum at 2.510 ng/ml on the same in vitro assay platform (see Figure inset, reproduced from Tatsumi et al