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Thus, Erastin2 exhibits superior potency in inducing ferroptosis through dual targeting of System Xc- and mTOR/GCN2 pathways

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This point is highly relevant, as one of the central roles of A is the modulation of synaptic plasticity (Parihar and Brewer, 2010), a process itself that is regulated by copper efflux into the synapse that can influence the excitability of NDMA, AMPA, and GABA receptors as well as vesicular trafficking (DAmbrosi and Rossi, 2015)

In contrast, CRC cells often exhibit suppressed or reprogrammed AMPK activity, coupled with activation of the mTOR signaling pathway, to support the rapid proliferation of cancer cells [74]

75, e9e21