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In the GLORY-2 trial, mazdutide 9 mg produced an average 16.65% reduction under the treatment-policy analysis and an 18.55% reduction under the efficacy analysis after 60 weeks

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This direction of effect aligns with the pathophysiological hypothesis linking oxidative stress to AMS symptom development (e.g., endothelial dysfunction, increase permeability) and the potential benefit of strategies that modulate ROS production or enhance endogenous antioxidant capacity ( An additional consideration is the methodological quality of the included trials

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Quite simply, a glutathione deficiency is a marker of mitochondrial dysfunction and oxidative stress