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These accumulated metabolites act as competitive inhibitors of nuclear -KG-dependent dioxygenases, including multiple histone demethylases and TET family DNA demethylases and resulting fundamentally alteration the cellular transcriptomic landscape (111,112)

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10.1038/s12276-020-00504-8 243 YuC.WangN.ChenX.JiangY.LuanY.QinW.et al (2023)

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However, the cardiovascular effects of these metabolites require evaluation in the context of host metabolic status, as some oxysterols may exert protective effects through anti-inflammatory mechanisms, while overall dysbiosis of gut microbial metabolic activity may drive harmful metabolite accumulation (Li et al., 2024)

& Mathew, S