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This discovery opens the door to potential norovirus treatment strategies that use mRNA technology to deliver protective IgA antibodies directly into the gut

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Mechanisms deciphered in these studies indicate that the interaction between serotonin and GLP-1 in the hindbrain increases proopiomelanocortin/cocaine- and amphetamine-regulated transcript (POMC/CART) neuron activity, leading to induction of satiety signals and suppression of neuropeptide Y/agouti-related peptide (NPY/AgRP) neuron activity, which in turn suppresses hunger

As a reminder, common GLP-1 peptides are not interchangeable on a milligram-to-milligram basis

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For those who have struggled with plateaus on existing medications, or who face multiple interconnected health issues, this kind of therapy may offer a more complete solution