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Ferroptosis and acute kidney injury (AKI) Ischemiareperfusion injury (IRI), rhabdomyolysis, and chemical drugs are common causes of AKI [134, 135]
[1] It functions as a dual glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide-1 (GLP-1) receptor agonist
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By this stage of life, the cumulative decline in glutathione production is more significant
Targeting neural reflex circuits in immunity to treat kidney disease
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