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Rojas-Ramos et al., 2003) coupled to the CCR3 receptor, while IL-4 and IL-3 favor the change of immunoglobulin (Ig) isotype in B cells, with the subsequent production of IgE (Figure 1)

In addition, bortezomib treatment can also lead to mitochondrial damage, causing mitochondria to produce a large amount of reactive oxygen species (ROS), thereby damaging mitochondrial function and producing cytotoxic effects [29]

These findings collectively point to Epitalon as a functional telomerase modulator, capable of extending telomeres, delaying replicative senescence, and maintaining cellular health across multiple biological systems