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This section examines how mitochondrial metabolism contributes to therapeutic resistance across chemotherapy, targeted therapy, and immunotherapy, and considers its potential as a clinically relevant target for intervention (Fig
Potent body weight loss, and therapeutic efficacy in a NASH animal model by a novel long-acting GLP-1/Glucagon/GIP tri-agonist (HM15211)
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F., Lingvay, I., Kushner, R
OeyNARuiterJPAtti-BitachTIjlstLWandersRJWijburgFA
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