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In preclinical models, BPC157 has been shown to modulate expression of key inflammatory mediators by downregulating proinflammatory transcription factors such as NFB and AP1 while upregulating antiinflammatory genes linked to IL10 signaling, thereby shifting the balance toward resolution of inflammation in chronically injured tissues (24,25)

This conclusion may re-focus attention on channel- and/or transporter-mediated contributions to efflux from neurons and influx into astrocytes (Benjamin, 1987)

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The accumulation of misfolded proteins within the ER lumen leads to osmotic imbalance, resulting in water efflux and expansion of the ER compartment

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