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These changes can cause oxidative modifications to reactive thiols of structurally- or functionally-critical proteins, such as transcription factors 43 , signal transduction molecules 44 or metabolic enzymes 45 , and thereby evoke cellular defense mechanisms against oxidative damage 46

As the understanding of ferroptosis and its mechanisms continues to improve, there is increasing evidence that members of the ubiquitin-proteasome system regulate ferroptosis and play an essential role (Figure 3)

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