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d , BN-PAGE immunoblot on isolated WAT mitochondria from Control mice at ZT2 and ZT14 using an antibody mix against OXPHOS complex subunits (n = 5)
43 This suggests that this mtDNA mutation is a risk factor that requires interactions with nuclear mutations, polymorphisms or epigenetics and/or environmental triggers or modulators in order for the disease phenotype to be expressed
Renal effects, including acute kidney injury, have been reported rarely in post-marketing surveillance, typically in the context of severe gastrointestinal adverse effects leading to dehydration
Peng, L
The former rate may seem especially impressive, but it is worth noting that this was reached only after 15 trials with some implicit training between each trial
J Clin Endocrinol Metab, 96(1), 150158
