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However, for people who are already vulnerable to disordered eating, the appetite suppression these medications produce can, in some circumstances, interact with or reinforce difficult patterns around food

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Figure 13 illustrates the effects of prenatal exposure to VPA, showing that HDAC inhibition may exacerbate oxidative stress by dysregulating the expression of genes involved in the antioxidant response, thereby promoting redox imbalance through increased ROS production and impaired antioxidant defenses (Gottlicher 2001

Glucagon-like peptide-2 increases intestinal lipid absorption and chylomicron production via CD36