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Therefore, reduced CI-driven NADH oxidation would constrain lipogenic capacity most prominently in this depot, which is consistent with our observations that impairment in CI reduces adipogenesis and promotes hypertrophic remodelling in gWAT

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How This Works IGF-1 LR3 is a synthetic analog of human insulin-like growth factor-1 engineered with an N-terminal extension (13 amino acids) and a glutamic acid substitution at position 3, resulting in significantly reduced binding affinity to IGF binding proteins [1]