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We previously demonstrated that RMC exhibits a hot immune-inflamed phenotype characterized by upregulation of immune checkpoints such as cytotoxic T-lymphocyte-associated protein 4 (CTLA-4) and programmed cell death protein 1 (PD-1) in the tumor immune microenvironment 10

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The emphasis on nimacimab's differentiation compared to existing treatments suggests that the companys prior developments may not have met market expectations or standards

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