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Reduced palmitoylation of endothelial nitric oxide synthase (eNOS) in insulin-deficient or insulin-resistant diabetic mice induces endothelial dysfunction manifested as inflammation and impaired angiogenesis (180)

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tumefaciens cultures carrying the VIGS vector targeting GFP as control (RNAi - Ctrl) or MEKK1 (RNAi -MEKK1 )