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Using an artificially assembled blood brain barrier consisting of endothelial and astrocyte layers, we found that labeled GLP-1 time-dependently crossed the barrier and the presence of GLP-1 receptor antagonist blunted this transit

In summary, our study demonstrates that pancreatic islet function is largely preserved in a mouse model of BTHS, despite marked cellular, morphological, transcriptional and metabolic adaptations

Neuroscientists describe this as a dampening of dopamine signalling across reward pathways

To learn more about Fibersol, download the white paper here: Satiety and GLP-1 White Paper Case Studies: Start-Your-Day-Right Breakfast Shake Power Packed Apple Cinnamon Bites Sources: 1 World Health Organization (2024) 2 IQVIA MIDAS, Jun 2023

1988;27 Suppl 1:86-93