Hypermetabolic state in the 7-month-old triple transgenic mouse model of Alzheimers disease and the effect of lipoic acid: a 13C-NMR study
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PMCID: PMC5295439
Glutathionylation mediates angiotensin II-induced eNOS uncoupling, amplifying NADPH oxidase-dependent endothelial dysfunction
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FACS analysis confirmed that at passage 10, 54.7% of EpCAM+ HBs co-expressed the proliferative marker Ki67, and immunostaining showed sustained co-expression of AFP, HNF4, and Ki67