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The increased bioavailability of NO through CIT supplementation serves to optimize microvascular recruitment and the transport of glucose and amino acids, thereby fostering a conducive environment for leucine-mediated activation of the mTOR and exercise-induced metabolic adaptations (82)
In PD patients, plasma ferritin is moderately positively correlated with the levels of the inflammatory cytokine IL-6, hepcidin, the main regulator of iron homeostasis, and with L-DOPA levels [67]
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