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Therefore, Ferroptosis can be considered as a byproduct of cellular metabolism

In a rotenone-induced PD model in rodents, DA4-JC also alleviated motor symptom dysfunctions, protecting dopaminergic neurons mediated through the restoration of mitochondrial function via the Akt/c-Jun N-terminal kinase (JNK) signaling pathway [116]

It catalyzes ROS generation that overwhelms cancer cell redox homeostasis (Jung et al., 2016)

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These findings emphasize the need for precision strategies that consider dose thresholds, exposure duration, and patient-specific pathology to effectively modulate LPSs net pathological effect in HF pathogenesis

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