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This phenomenon may be due to saturation of L-carnitine transport into skeletal muscle at physiological L-carnitine plasma concentrations (4060 mol/l) and/or by the significantly higher carnitine concentration in skeletal muscle relative to plasma, rendering passive transport impossible [24]
By contrast, the expression levels of these key autophagy genes in siATF4 transfected HuOCSCs treated with anisomycin were significantly lower than those in the control group (Figure 6B)
Distinct roles of the methylcytosine oxidases Tet1 and Tet2 in mouse embryonic stem cells
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