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The results of this study showed that viruses lacking other IE viral proteins (ICP22 or ICP47) activated p38 and JNK similar to the wild-type virus, suggesting that these proteins are not necessary for p38 and JNK activation (Hargett et al., 2005)

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Following treatment, PSA slopes from 6 months to 8 months persisted at the same levels in the two arms ( 3.4.2.3 Effect of SFN on histological and gene expression findings Several results on the expression of important genes and immunohistochemical markers were obtained from the included studies on three cancer types: PCa, breast cancer, and melanoma ( In PCa, the main investigations were carried out on the expression of important proteins involved in PCa etiology, including AMACR, ARLNC1, epigenetic histone modifications, histone deacetylases (HDAC), Ki67 nuclear antigen, cyclin-dependent kinase inhibitors, nuclear factor-erythroid factor 2-related factor 2 (Nrf2), epidermal growth factor (EGF) ( In total, 40 differentially expressed genes correlated with SFN treatment were identified in one study, including the downregulation of two genes previously implicated in PCa development (i.e

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3 Monitor outcomes such as pain scores, analgesic consumption, time to functional milestones, wound/repair integrity, and incidence of complications (e.g., delayed wound healing, infection)