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We also express our sincere gratitude to the administrative support of the Chang Gung Memorial Hospital Clinical Trial Center and all staff of the Taiwan Clinical Trial Consortium (TCTC)

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In particular, the animal study 7 in which pharmacological inhibition of GPx activity significantly reduced brain infarct damage suggested that under reduced oxygen supply, H 2 O 2 may exert either physiological or protective roles in the brain through its metabolic degradation to oxygen by catalase compensating for the lack of oxygen that occurs in the tissue after an ischemic event