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In schizophrenia, glutamate hypofunction, particularly in the prefrontal cortex, is believed to contribute to cognitive and negative symptoms, while overactivity of glutamate transmission may exacerbate positive symptoms such as hallucinations

1), and these variables were included in Model A

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External resources National Institutes of Health: CXXC5 Mediates DHT-Induced Androgenetic Alopecia via PGD2 MDPI Cells: CXXC5, PGD2, and Wnt/Beta-Catenin Pathway in Alopecia Follicle Thought: PTD-DBM Update Q&A with Dr