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Neurons were treated with a vehicle, Nle-AngIV, or Dihexa for 5 days or acutely for 30 minutes
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the D-substitutions make GHRP-2 substantially more resistant to enzymatic degradation than a natural peptide of the same length would be
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It binds poorly to IGF-binding proteins, which normally sequester IGF-1 in the body, allowing it to remain active and available for a significantly longer period
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