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This condition has been observed to have a familial tendency (16)
Braunstein GD et al
6,7 GLP-1 was later identified in the 1980s as another primary incretin hormone responsible for the stimulation of insulin release to cover carbohydrate utilization following a meal
GLP1R variant is associated with response to exenatide in overweight Chinese Type 2 diabetes patients
This is less common with liposomal forms, as the nutrient is protected and absorbed more efficiently
In support to this theory, it has been demonstrated that GIP infusion increases the proliferation of neuronal progenitors in the dentate gyrus, while mice lacking GIP displayed deterioration in memorial tasks as a result of reduced neurogenesis [24]
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