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Interestingly, GLP-1 is also known to decrease the rewarding effects of food intake by interacting with parts of the mesolimbic dopamine system such as the ventral tegmental area (VTA), nucleus accumbens (NAc) [34], and also GABAergic circuits in the laterodorsal tegmental nucleus (LDTg) [35], which makes brain GLP-1R signaling a promising target for the treatment of alcohol and cocaine abuse disorders [36, 37]
Unlike many therapeutic peptides that require protective carriers to survive in the body, BPC-157 maintains its structure long enough to reach target tissues and initiate its effects
J Mol Struct 875:3241
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Some pages also point to this study on bacteria : erc.europa.eu/projects-figu
