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In this context, cytotoxic T-lymphocytes (CD8+) are the main cells implicated in disease pathogenesis.78 However, although antibodies reactive against melanocytes (e.g., anti-MelanA, anti-MCHR1, anti-tyrosinase, anti-gp100, and anti-tyrosine hydroxylase) have elevated serum titers in patients with vitiligo, they do not correlate with disease activity.79 Nevertheless, the literature is controversial, and the exact role of anti-melanocyte antibodies in vitiligo remains unclear

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doi: 10.1017/S0967199409990311 116 PolakGRolaRGogaczMKozio-MontewkaMKotarskiJ

Less necrosis developed in treated wounds, suggesting the peptide helped preserve marginally viable tissue that might otherwise have died