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In contrast, its oxidized form (GSSG) is a byproduct produced when GSH neutralizes harmful compounds

[PMID: 9626749] PubMed record related to this compound for laboratory literature review

Cystic fibrosis, usually due to severe mutation in bothalleles of the cystic fibrosis transmembrane conductance regulator ( CFTR ) gene, causes an accelerated non-calcific form of chronic pancreatitis that begins in utero : oxidative stress and inflammation are now regarded as integral features of the disease, driven by unfolded CFTR via the ER stress-UPR system (121, 177)

For these reasons, a Y250-containing site is likely to be less sensitive to the impacts of hypothetical chemical modification/remodeling of the disulfide bonds mentioned

These new findings encourage a novel working hypothesis for further research, that a chemical-modification mechanism enables transformation of HGF through interaction or reaction with LASSS under physiological conditions, resulting in the retention of HGFs biological activity after exposure to peroxynitrite, at least until an enhanced form of HGF undergoes metabolic turnover in vivo

I'm excited about our small-scale clinical trials