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Under the stimulation of lactate, the METTL3/YTHDC1 axis mediates the stability of ACSL4 mRNA through m6A modification, which leads to an increase in ACSL4, inducing ROS accumulation and mitochondrial-dependent ferroptosis, thereby exacerbating lactate-induced ferroptosis in pulmonary epithelial cells[209]

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Factors such as diet, age, sex, and metabolic status have not been fully controlled

Practitioners should provide comprehensive post-treatment care guidelines, ensuring patients are well-informed and prepared to manage any mild discomfort