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The overexpressed GSH, in the form of cellular free thiols, can generate highly reductive TME to resist DNA damage and protein homeostasis disorder, and protect tumor cells from programmed cell death (Labarrere and Kassab, 2022)
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Oncology effects are highly context dependent
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Autologous lipofilling: coenzyme Q10 can rescue adipocytes from stress-induced apoptotic death
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