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Early vascular remodeling is an adaptive protective mechanism, and vascular remodeling during decompensation can promote the progression of atherosclerosis and the occurrence of complications

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78 However, the demonstration of endothelial dysfunction in the presence of increased expression of eNOS indicates that the capacity of the enzyme to produce NO may be limited, and the concept that eNOS itself can be a superoxide source and thereby contributes to endothelial dysfunction provides a further explanation to this observation

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